This Paper investigates further the new Principles regulating the relations as between extension perception and division of cells
Further:
Returning to the original Equation: Principle above-mentioned:
The rate extension of cells will be as directly proportional:equivalent:identical to the rate of perception of the same divided by the rate of division.
Now in cells - in the history of any cell - there might be relatively high levels of extension:perception:division and relatively low levels of the same (perception:extension:division).
(All these levels are relative; all this is relative)
Thus the Principle:
The rate of level of extension:perception:division in cells will be as directly proportional to the rate of level of propensity to cell division
And vice-versa
And further:
The rate of propensity to cell:division will be as directly proportional to the rate of propensity to cell:extension
And vice-versa
Note the import of the Vice-versa here.
Therefore - and as by syllogistic reasoning - :
The rate of level of extension:perception:division in cells will be as directly proportional to the rate of propensity to cell:extension
And vice-versa
Now it is manifest that for cells to divide there must be relatively high levels of perceptions and relatively high levels of extensions
For there can occur the case where P being divided by E to obtain D if the rate of E is very low and the rate of P very modest (and thus not high) - logically and in the ideal state - a high rate of D - and therefore of cell:division will apply.
But this is not so.
For if in the Equation:Principle:Formula D = P divided by E both P and E are low their division is also low - and the rate of propensity to cell-division will be low equally and in as direct proportion.
But if a high rate of P is divided by a high rate of E then the resultant will be a high rate of D (all are relative)
And if there be an ever-increasing (continuous) rate of (increase of) P as well as of E them there will be - and in as direct proportion - an increasingly (continuously increasing) high rate of D.
In the equation P = E X D (and its algebraic transformations namely D = P divided by E and E = P divided by D) - E and D are transmitted: hereditary and P is induced: bombarded
What is hereditary transmitted ::: set:fixed
What is induced:bombarded is in > evolution:progression
Therefore :
Out of E, D and P:
only P can be changed to > P
If P is changed to > P
Then D = > P divided by E
· D
And:
E = > P divided by D
· E
Thus in division of cancer-cells since P is changed to > P therefore and as above manifested E will change to > E and D will change to > D
Whilst in “normal” cell-division E, P and D remain as they are.
Therefore the genesis of the propensity to cancerous growth is the changing of P to > P
And the engineering means?
The direction mode and extent are manifested by the above.
1/9/1996
8.15 A M
Further:
Returning to the original Equation: Principle above-mentioned:
The rate extension of cells will be as directly proportional:equivalent:identical to the rate of perception of the same divided by the rate of division.
Now in cells - in the history of any cell - there might be relatively high levels of extension:perception:division and relatively low levels of the same (perception:extension:division).
(All these levels are relative; all this is relative)
Thus the Principle:
The rate of level of extension:perception:division in cells will be as directly proportional to the rate of level of propensity to cell division
And vice-versa
And further:
The rate of propensity to cell:division will be as directly proportional to the rate of propensity to cell:extension
And vice-versa
Note the import of the Vice-versa here.
Therefore - and as by syllogistic reasoning - :
The rate of level of extension:perception:division in cells will be as directly proportional to the rate of propensity to cell:extension
And vice-versa
Now it is manifest that for cells to divide there must be relatively high levels of perceptions and relatively high levels of extensions
For there can occur the case where P being divided by E to obtain D if the rate of E is very low and the rate of P very modest (and thus not high) - logically and in the ideal state - a high rate of D - and therefore of cell:division will apply.
But this is not so.
For if in the Equation:Principle:Formula D = P divided by E both P and E are low their division is also low - and the rate of propensity to cell-division will be low equally and in as direct proportion.
But if a high rate of P is divided by a high rate of E then the resultant will be a high rate of D (all are relative)
And if there be an ever-increasing (continuous) rate of (increase of) P as well as of E them there will be - and in as direct proportion - an increasingly (continuously increasing) high rate of D.
In the equation P = E X D (and its algebraic transformations namely D = P divided by E and E = P divided by D) - E and D are transmitted: hereditary and P is induced: bombarded
What is hereditary transmitted ::: set:fixed
What is induced:bombarded is in > evolution:progression
Therefore :
Out of E, D and P:
only P can be changed to > P
If P is changed to > P
Then D = > P divided by E
· D
And:
E = > P divided by D
· E
Thus in division of cancer-cells since P is changed to > P therefore and as above manifested E will change to > E and D will change to > D
Whilst in “normal” cell-division E, P and D remain as they are.
Therefore the genesis of the propensity to cancerous growth is the changing of P to > P
And the engineering means?
The direction mode and extent are manifested by the above.
1/9/1996
8.15 A M